Research and regulatory reference

ACE-031

A source-linked reference on the identity and evidence context of ACE-031. Sources checked 6 September 2026.

Source and review status

Published sources checked

Sources checked on: 6 September 2026

Qualified clinical review: Pending. This page has not been clinically reviewed.

About this source check

AI-assisted editorial source checking covers the cited material and its stated limits. It is not a systematic review, verification of a supplied product, or clinical sign-off.

Editorial role: InfoAddict. Last material change: 6 September 2026. Next source review: 6 December 2026.

Identity

ACE-031 is an activin-receptor-IIB and IgG1-Fc fusion protein.

Sources: Published 2017

A named substance and a finished product are different claims. The name does not verify formulation, concentration, purity or equivalence to material used in a study.

Research context

The trial was stopped after its second dosing regimen because of potential safety concerns including epistaxis and telangiectasias. Its reported trends did not establish a treatment effect.

Sources: Published 2017

The source documents a specific identity or research setting. It does not independently verify a commercial product, establish product equivalence or answer a person-specific health question.

Evidence boundary

This entry reports the limited source context above. It does not make a personal treatment recommendation or infer a result from a different molecule, formulation, model or study population.

The appropriate conclusion is limited to the cited record, not to untested products or uses.

Published sources

  1. Myostatin inhibitor ACE-031 treatment of ambulatory boys with Duchenne muscular dystrophy: Results of a randomized, placebo-controlled clinical trial

    Published 2017. Current article record; accessed 6 September 2026.

    Supports: A randomized, placebo-controlled study in ambulatory boys with Duchenne muscular dystrophy.

Change history

DateChangeClinical-review status
6 September 2026Source-linked reference publishedPending

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